The disease is expressed by almost constant pain, with a change in their intensity. Depending on the type of lesion, thoracic or abdominal aorta, there may be: pain in the chest (can be burning, pressing, baking, sharp); the occurrence of tachycardia and shortness of breath; pain in the peritoneum and / or lower back; frequent cough; irradiation of pain in the neck, shoulder blades, abdomen, arms; pulse asymmetry (differences between the two sides of the radial or carotid arteries); systolic murmur over a pathologically altered area of Prednisolone; chilliness and weakness all over the body; cold extremities due to circulatory disorders.
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Aortitis is a disease of the walls of the aorta, characterized by inflammation throughout its thickness or a separate part. Sometimes the disease proceeds for a long time without any symptoms. The main cause of inflammation of the aorta is an infectious lesion of its walls. The diagnosis is made through clinical, laboratory and instrumental studies. Aortitis is a dangerous disease, untimely treatment of Prednisone can lead to death.
Causes and predisposing factors. The main cause of aortitis is an infection in the aortic wall, which can be streptococcal, tuberculous, or syphilitic. Men and women can be affected equally. Also, the reasons may be: lung abscess; aortic aneurysm; allergic lesions of the aorta; brucellosis; inflammation in cells and tissues; injury; syphilis; damage to the muscle membrane; rheumatic fever.
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Sometimes thromboangiitis obliterans, ankylosing spondylitis becomes a predisposing factor in the development of aortitis. It can be expressed by systemic collagenoses. Also, the disease can occur after infection with malaria or gonorrhea. In addition to the aorta, when affected by pathogens, the orifices of Prednisolone arteries, the valve, and adjacent tissues may also be involved in the pathological process. At the same time, changes are taking place with the interior lining. Due to deformation, it experiences wall thickening, as well as sclerosis. Connective tissue replaces the lost elastic components of the aortic walls. A so-called aneurysmal sac is formed. In advanced states of the disease, such a bag can burst.
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Patients with giant cell arteritis, psoriatic arthritis, deep mycoses, Takayasu's disease are also at risk. With this disease, the thoracic aorta is most often affected, but there are cases of damage to its abdominal part. The division into forms occurs depending on the causes that caused the disease. It could be infectious or allergic aortitis.
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Infectious is subdivided into syphilitic or nonspecific infectious. Allergic to - toxic-allergic, autoimmune, infectious-allergic. Aortitis is also subdivided according to the nature of the course of the disease - acute, subacute, chronic. Acute is divided into purulent or necrotic. The spread of the disease is divided into ascending, descending, diffuse.
This is a special form of aortitis that develops on the basis of some pathologies, but not against the background of an infectious lesion. Sometimes in the wall of the aorta develop necrosis such as microinfarcts. The lesion may begin with the ascending aorta, spreading to its branches. Then Takayasu's disease develops.
Violation of blood circulation occurs mainly in the branches of the aorta, which come from the pathological site. As a result, ischemia and hypoxia occur in the tissues supplied by them, parts of organs.
The main and most severe complication of the disease is the occurrence of an aortic aneurysm, which threatens its exfoliation, and subsequently - rupture and death for the patient.
There are several specific symptoms of aortitis. This applies to syphilitic, infectiouso nonspecific, tuberculous aortitis and aortitis that accompanies Takayasu's disease. Syphilitic aortitis. The anamnesis is specific, the symptoms do not make themselves felt for a long time (from 4-5 years to 23 years or more from the moment of infection of the patient). The main complication is an aneurysm. Nonspecific infectious aortitis.
The onset of aortitis is very rapid and abrupt, often after another acute infectious disease. Accompanied by fever of unknown etiology. The main provoking factor is Staphylococcus aureus. Tuberculous aortitis. The patient has a history of pulmonary tuberculosis.